By Mieczyslaw Pokorski (eds.)
A host of neurotransmitters and neuroactive ingredients underlies breathing law in overall healthiness and disorder. the center piece of investigations relating to variation to hypoxia and sensorial conception has been the dopaminergic method. it really is now transparent complicated interplay between a variety of neuroactive components, instead of a unmarried one, types the root of breathing adjustments. The learn on neurotransmitter interactions offers the data of ways the mind features and a brand new point of realizing of mind-to-body connection, which opens up avenues for novel healing interventions.
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Extra resources for Neurotransmitter Interactions and Cognitive Function
We have also recently reported that GUO administered intraperitoneally to rats is able to pass the blood-brain barrier (Giuliani et al. 2012a) and stimulates the release of neurotrophic factors (Di Iorio et al. 2004). P. Giuliani et al. Taken together the present findings demonstrate that GUO might be of research interest as a novel therapeutic approach for treatment of PD. Acknowledgments PG and PB contributed to this work equally. Conflicts of Interest The authors declare no conflicts of interest in realtion to this article.
The real-time monitoring of VOCs enables to assess both the effect of olfactory stressor and the level of central neural fatigue in both healthy and sick subjects in combination with cognitive performance (Mazzatenta et al. 2013b, c). Here, we applied the breath real-time monitoring of VOCs to investigate the chemo-responsiveness in the long-term irreversible post-traumatic anosmia. 1 Case Presentation An anosmic Caucasian male patient, aged 62, volunteered for the study. The patient provided written informed consent.
In vitro treatment of cells with 6-hydroxydopamine (6-OHDA) and in vivo administration of this neurotoxin to laboratory animals represent one of the most used experimental models of PD. Moreover, 6-OHDA content is increased in the brain and urine of patients with PD treated with L-DOPA (Andrew et al. 1993; Curtius et al. 1974) and a role for this neurotoxin in the pathogenesis of the disease has been suggested. We have recently reported that guanosine (GUO) protected SH-SY5Y neuroblastoma cells when exposed to neurotoxins, including MPP+ and 6-OHDA (Giuliani et al.